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MCT1 inhibition compels cancer cells to compete for glucose, thereby inducing apoptosis in hypoxic cancer cells, while MCT4 inhibition triggers intracellular acidosis under hypoxic conditions (142)
indicated that GPX4 and GSH levels were decreased, and cellular iron and MDA levels were increased in a mouse model of myocardial ischemiareperfusion (MIR, ischemia 30 min and reperfusion 24 h) injury [90]
Glutamate can be produced by either GOGAT or GDH and glutamine is produced by glutamine synthetase via the GS/GOGAT cycle
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