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Through synergistic crosstalk with the Nrf2 pathway, FXR activation mediates: Metabolic dysregulation amelioration, Oxidative stress and inflammation attenuation, Myocardial fibrosis and cardiac dysfunction suppression (28)
Contributing factors include: reduced nutritional intake (proteins, essential amino acids, competition between amino acid transporters, vitamin insufficiency [riboflavin: GSSG reductase, nicotinic acid: NADPH, pyridoxine (B 6 ): SAMe pathway], mitochondrial dysfunction: ATP deficiency, down regulation SAMe synthase, oxidation of SAMe pathway enzymes, enhanced GSH utilization: conjugation reactions (toxins, drugs), and overwhelming oxidative challenge
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