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Other reported mechanisms include activation of FAK-paxillin complexes (which help cells migrate and attach to surfaces during wound healing), JAK-2 signaling, and ERK1/2 pathway activity (Sikiric et al., Current Neuropharmacology, 2016
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If you read those statistics for any other disease, it would be a catastrophe
[18] [19] Clinic data also note it does not disrupt hormonal pathways targeted by finasteride, making it attractive for users sensitive to hormone-based treatments
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