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Experimental investigations have reported interactions with VEGFR2 phosphorylation, focal adhesion kinase (FAK) activation, ERK1/2 signaling, nitric oxide-associated pathways, and extracellular matrix regulatory networks

Egecioglu E, Engel JA, Jerlhag E

151 Findings from cellular studies with Caco-2 cell monolayers (a common model for studying intestinal permeability) also suggest collagen peptides reduce intestinal permeability induced by the inflammatory trigger TNF-