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iNKT cell modulation, AMPK phosphorylation, IL-23/Th17 regulation, and reduction of T cells have all shown to be possible mechanisms by which GLP-1RAs exert anti-inflammatory effects
Receptor binding and mechanism of action The main signaling mechanism of GLP-1 is the activation of adenylate cyclase via the Gs subunit, leading to increased production of cyclic AMP [6, 10]
2 The reduction may be greater in people with higher starting blood pressure
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