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The peptide binds to vascular endothelial growth factor receptor 2 (VEGFR2), triggering Akt-eNOS signaling that increases nitric oxide production in blood vessel cells
Interventions in this direction mainly manifest in two complementary logics: one is to indirectly block hypoxia-induced pro-oxidant signaling axes by inhibiting HIF function [486]
88 TakahashiJ
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