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GHK-Cu has demonstrated the ability to suppress excessive inflammatory signaling and reduce leukocyte infiltration in tissue injury models, indicating regulatory effects on immune-cell migration and local inflammatory cell density (31,32)
11,12 TLR3, TLR7, and TLR9 recognize nucleic acids from damaged cells, while proteins released from cells and ECM components post-tissue injury can engage TLR2 and TLR4
28 In addition, homocysteine is known to cause endothelial (protective lining) damage, a recognized contributor to the progressive loss of kidney function
Chen SJ, Kuo CH, Kuo HC, et al
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