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Genotyping of SOD1 G93A transgenic mice was carried out as previously described 23
Seeley, University of Michigan) and Wnt1-Cre2 mice were bred at Mount Sinai Hospital (Toronto, Canada), as previously described 19,20 , to produce a CNS knockdown of the Glp1r ( Glp1r Wnt1/ )
The question that triggered this paper The turning point was very simple: If glucagon is essential for stress adaptation, amino-acid clearance, and hepatic energy mobilization, what happens when we chronically silence it
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