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Given that Bacteroides abundance is typically reduced in patients with COPD, particularly during acute exacerbations (Bowerman et al., 2020), these findings imply that restoration or targeted modulation of this genus could constitute a plausible mechanism by which microbiota-based interventions augment Trp metabolic capacity and regulate disease progression

This function is referred to as reverse cholesterol transport (RCT)

1 Introduction Cancer fundamentally stems from the disruption of normal cellular growth and division, highlighting the necessity for effective therapeutic strategies to address this disease (Zhang et al., 2022)

Therefore, a comprehensive understanding of these compensatory mechanisms is imperative for the development of more effective therapeutic strategies that target cancer cells [212]