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10.4049/jimmunol.173.1.531 91 McLaneM
A mitochondria-targeted covalent organic framework (COF) prodrug that is selectively activated by high intramitochondrial GSH levels was shown to trigger catalase collapse within the organelle, thereby amplifying local chemodynamic therapy (CDT) through enhanced ROS accumulation and mitochondrial dysfunction
Characterization of the human class Mu glutathione S-transferase gene cluster and the GSTM1 deletion
Effects of thalidomide on Th17, Treg cells and TGF-beta1/Smad3 pathway in a mouse model of systemic sclerosis
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