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Ferroptosis has emerged as a potential key player in various diseases and the initiation of organ fibrosis
Interestingly, research has shown that even in animals lacking beta-3 adrenergic receptors, acute AOD-9604 administration still increased energy expenditure and fat oxidation, suggesting additional metabolic pathways may be involved beyond the primary beta-3 receptor mechanism
Thus, there is some promise in improving blood glutathione status through supplementation [16, 17], however, there is a need to explore ways to optimize this effect
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