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Its strong copper coordination is thought to improve its pharmacokinetic profile by allowing slow, localized action rather than broad systemic exposure

What this data doesn't tell us: whether NNMT suppression sustained beyond 12 weeks alters NAD+ metabolism in ways that affect mitochondrial function, whether methylation pathway compensation occurs over longer timescales, or whether adipose tissue remodeling continues linearly or plateaus

Future studies need to clarify the changes in ASC subtypes during aging and determine the possibility of targeting markers of these ASCs to reverse the aging effects of adipose tissue