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NAC is a direct precursor to cysteine, the rate-limiting amino acid in glutathione synthesis, and it reliably increases intracellular GSH levels across multiple tissues
This recognition step triggers further cytokine release and inflammation, thereby perpetuating an inflammationferroptosisinflammation positive feedback loop (138)
Pharmacological inhibition of the PI3K/mTOR pathway abolished such effects, suggesting that the metabolic actions of GLP-1 depend on mTOR activity
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