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By inhibiting NNMT, which preserves SAM and NAD+ pools, supports sirtuin activity, and reduces adipocyte lipogenesis

Elevated BHB levels suppressed AKT activation in cardiomyocytes, while BDH1 overexpression effectively restored AKT/GSK3 pathway activity and ameliorated BHB-induced mitochondrial dysfunction and cardiomyocytes apoptosis

Reduced PfK13 levels were linked to impaired hemoglobin digestion, decreased free heme levels, and consequently, decreased artemisinin activation

Caspase-independent cell death is involved in the negative effect of EGF receptor inhibitors on cisplatin in non-small cell lung cancer cells