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-arrestin-1 maximal responses closely mirrored those of -arrestin-2 (Supplementary Fig
[30] Release of GSTs as an indication of organ damage [edit] The high intracellular concentrations of GSTs coupled with their cell-specific cellular distribution allows them to function as biomarkers for localising and monitoring injury to defined cell types
One key pharmacologic distinction matters for hepatic impairment specifically
The aims of these trials is to achieve fat-selective weight loss, improved body composition and end-organ protection via a triple mechanism effect in people living with obesity [114]
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