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In essence, it functions as a signaling molecule: when introduced into damaged tissue, it appears to activate multiple healing pathways that help the body repair itself more efficiently

Importantly, pre-administration of LASSS to mice prevented the disuse-induced HGF nitration observed in a tail-suspension model for muscle atrophy, while GSSSG did not

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Studies suggest this rebalancing occurs through effects on thymic peptide signaling and restoration of neuroendocrine-immune system communication that deteriorates with aging