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Mechanisms underlying the action of TG include both genomic (via nuclear receptors) and non-genomic (membrane) effects
White JR
Gallic acid attenuates cerebral ischemia/re-perfusion-induced blood-brain barrier injury by modifying polarization of microglia
Within this context, Melanotan 1 has been examined as a synthetic agonist that might modulate MC1R signaling and elevate cAMP dependent responses, including pathways associated with melanogenesis and genomic maintenance
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