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Although GIP receptor activation may exert anti-emetic effects through brainstem modulation, these effects appear insufficient to completely counterbalance GLP-1 mediated emesis during early treatment and dose escalation ( In contrast, glucagon receptor activation is independently associated with nausea and vomiting through both central and peripheral mechanisms, including effects on gastric motility and autonomic signaling
This reduces the risk of diabetic ketoacidosis, and you should contact your healthcare team for guidance on when to restart treatment
Ongoing check-ins help ensure youre responding well and that adjustments are made as needed
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