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They found that BPC-157 enhanced the outgrowth of tendon fibroblasts from explants, increased their survival under oxidative stress (HO challenge), and promoted FAK-paxillin phosphorylation, collectively suggesting a direct effect on the key cell type involved in tendon repair

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However, further assessment of P2X4 receptor behavior in microglia within the thalamus could provide additional insight into its role in enhancing fibromyalgia-related pain mechanisms via IL- 1

Presented for research literature review only Khoo TK et al