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Overall, constitutive autophagy maintains cardiomyocyte size, architecture, and basal performance in the healthy heart, whereas in failing myocardium, enhanced autophagy functions predominantly as a compensatory survival mechanism

Our comprehensive analysis of stromal compartments underscores mitochondrial metabolic dysfunction in ABMR, emphasizing that the mechanisms underlying oxidative phosphorylation downregulation are highly cell type specific

A combination of promoter methylation tests for FKBP6, INTS1, and ZNF516 can be detected early in blood cfDNA and urine ( MEG3 (Zhang et al., 2017) and SIM1 (Kim et al., 2018) may serve as potential plasma biomarkers for CC

Hepatic fat mobilisation resumes at the next scheduled dose