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Morton, and W
Adding a glucagon-receptor component to an incretin agonist is not an obvious move, and the published rationale rests on the observation that glucagon-receptor activity contributes energy-expenditure and hepatic effects that are separable from its effect on circulating glucose, which the concurrent GLP-1-receptor component is positioned to offset
(Wolffenbuttel BHR, Wouters HJCM, Heiner-Fokkema MR, van der Klauw MM
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