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Cocaine neurotoxicity and neurological damage stem from several mechanisms, including mitochondrial dysfunction (Cunha-Oliveira et al., 2010), oxidative stress (Poon et al., 2007), neuroinflammation (Correia et al., 2020), excitotoxicity (Moran et al., 2005), and autophagy (Cao et al., 2017)

This doesnt mean complete immobilization, which can impair tendon healing, but rather smart activity modification

Systemic and topical administration work through different pathways, potentially complementing each other

This particularly affects users with thicker skin or compromised barrier function that reduces penetration efficiency