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This promotes M2 polarization in the ischemic penumbra, attenuating blood-brain barrier disruption and neuronal apoptosis, a mechanism correlated with favorable clinical outcomes (248)

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This localized depletion is strongly correlated with increased ventricular lactate, pointing to severe neuroinflammation, an overwhelmed central nervous system antioxidant network, and a direct physiological explanation for the profound cognitive dysfunction and "brain fog" experienced by patients