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Indeed, cytosolic thioredoxin oxidation under acute H 2 O 2 stress was also shown to be dependent on the presence of cytosolic peroxiredoxins (Day et al , 2012)
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It is driven by iron-dependent phospholipid peroxidation and regulated by several cellular metabolic events, including redox homeostasis, iron handling, mitochondrial activity, and metabolism of amino acids, lipids and sugars [67]
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