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While oral glutathione supplementation can be effective in increasing glutathione levels, its absorption and bioavailability may be limited due to the digestive process
doi:10.1111/j.1445-5994.1989.tb00349.x
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Its binding constant (pKa 16.44) is similar to albumin's copper transport site (log = 16.2), allowing GHK to exchange copper with albumin for intracellular delivery.[3] Discovery and design rationale GHK was first identified in 1973 in fractions of human plasma that promoted survival of liver cells in culture
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