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Chronic liver injury induces metabolic adaptations in macrophages, characterized by aberrant glucose and lipid metabolism along with activation of stress responses such as ERS and autophagy, thereby promoting their transition toward inflammatory and fibrotic phenotypes (68, 172)
Rajman, L., et al
meningitidis can trigger rapid redistribution and degradation of cytosolic ferritin within infected epithelial cells and that cytosolic ferritin is aggregated and recruited to intracellular meningococci (Larson et al., 2004)
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