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However, excessive ROS accumulation or severe mitochondrial damage can shift their role toward tumor suppression
Besides, GLP-1RAs might be driven by anti-inflammatory mechanisms to inhibit the formation of macrophage foam cells and thus slow down the pathological processes of AS (125, 126)
Beyond taxonomic shifts, these microbial changes lead to functional alterations in microbial metabolic activity, resulting in modified production of bioactive metabolites that can influence multiple host physiological systems, including the gutovary axis (73, 74)
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