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[13] A further complication of severe deficiency is the neurological complex known as subacute combined degeneration of spinal cord
Our mechanisms emphasize the remodeling of the TME through a dual-pronged approach: reversal of T cell exhaustion alongside augmented recruitment, synergistically bolstering the efficacy of PD-1 blockade
High NNMT, as often seen in obesity, may be associated with marks favouring fat storage
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