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Jump to Figure 6 Figure 7: Development of GBB-adducts 158a and 158b as PD-L1 antagonists

Bax, a proapoptotic protein, increases mitochondrial membrane permeability and facilitates the release of caspase-9 and caspase-3

In a rat model of migraine, trigeminal neurons have been shown to have abnormal mitochondrial biogenesis capacity represented by the decreased mitochondrial DNA number of copies as well as the altered mRNA levels of peroxisome proliferator-activated receptor-gamma coactivator-1a (31), an essential regulatory factor in mitochondrial biogenesis (32)

A genome-wide haploid genetic screen identifies regulators of glutathione abundance and ferroptosis sensitivity