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However, under pathophysiological conditions, especially during APAP overdose, excessive NAPQI accumulates and binds to cellular proteins, also triggering oxidative stress, leading to hepatocellular damage and the release of damage-associated molecular patterns (DAMPs), contributing to further liver injury and inflammation
Activated T cells without GPX4 accumulate cellular lipid peroxides and undergo ferroptosis (21, 39, 40), while ferroptotic oxidative signals are produced primarily by iron-mediated Fenton reaction or enzymatic reaction via lipoxygenases or the dysregulated GSH antioxidant system (41, 42)
Kauvar says, but adds that there is no evidence to date of any toxicity at the concentrations currently used
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