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Progress may be gradual and difficult to perceive day-to-day but becomes obvious when comparing function before and after a full protocol cycle
MOTS-c interacts with Nrf2, enabling Nrf2 to recognize and bind to antioxidant response elements (ARE), which initiates the transcription of target genes and directly regulates ARE-containing target genes in the nuclear genome 61,62
Hepatitis C virus-induced up-regulation of microRNA-155 promotes hepatocarcinogenesis by activating Wnt signaling
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