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BPC-157's tendon-repair work (PMID 21030672) connects to TB-500's parent cardiac and corneal repair findings (PMID 15565145), while GHK-Cu's broader regenerative-research framework (PMID 26236730) and KPV's PepT1-mediated anti-inflammatory work (PMID 18061177, PMID 27458604) add complementary mechanism contributions
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APE1 co-precipitates more with nucleophosmin (NPM1) in C9orf72 patient tissue lysates compared to controls (Farg et al., 2017), and overexpression of NPM1 inhibits apoptosis in neuronal cells expressing poly GR 100 and poly PR 100 (Farg et al., 2017)
