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The initial pivotal studies that led to the first wave of GLP-1 RAs demonstrated that they work in several ways, including stimulating insulin release, slowing digestion, reducing appetite, and inhibiting glucagon release
In rodent primary chondrocytes, Liraglutide inhibited AGEs- or IL-1-induced expression and release of pro-inflammatory cytokines, including iNOS, TNF- and IL-6 [95,98]
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