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However, it is now believed that while cell senescence initially acts as a robust barrier against tumor initiation, the subsequent accumulation of senescent cells can paradoxically promote cancer recurrence and cause damage to neighboring tissues

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By reducing this fat, GLP-1 drugs may lower systemic inflammation, creating an anti-inflammatory shift

When combined theoretically, NAD could potentially support the metabolic improvements semaglutide initiates, though this represents extrapolation rather than proven synergy