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The hypothesis that deficient serotonergic neurotransmission contributes to the pathogenesis of MDD originated from clinical observations that amine-depleting drugs induced depressive states, whereas monoamine oxidase inhibitors, which prevent the breakdown of serotonin (5-HT), alleviated depressive symptoms [4, 5]

Western replications are sparse, and no Phase III trial has been completed outside Russia

The root meristemless1/cadmium sensitive2 gene defines a glutathione-dependent pathway involved in initiation and maintenance of cell division during postembryonic root development

The reasons are biological, not cosmetic