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We have produced a large-scale computational biology analysis of the SNO-proteome in the cortex of Shank3 mutant mice (a popular model of ASD [171, 282, 283]) and found that among the NO-related molecular alterations, the most prominent change was the activation of the mTOR signaling pathway [171, 176]
In addition, compared with normal individuals, the expression level of HNF4 was decreased at different stages in patients with HCC (Fig
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