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A reduction in experimental lesions may indicate cytoprotection-associated activity in a controlled model, but it does not necessarily explain whether the primary driver is endothelial preservation, anti-inflammatory signaling, epithelial restitution, altered motility, or a combination of these processes

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Due to its sleep-related synthesis, the peptide has attracted attention for its potential role in regulating sleep patterns and promoting sleep-related well-being [1]

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