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He graduated from the Sackler School of Medicine and completed training at the Robert Cooke Allergy Institute in New York City

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observed abundant autophagic vesicles and enhanced autophagic flux in SCs from the HG group (90), autophagy hyperactivity was also observed in the streptozotocin (STZ)-induced rat model of DPN in Wang et al., these effects were also found to potentially increase apoptosis in SCs and demyelination of sciatic nerves in rats with DPN (91)

Insufficient progesterone production from CL dysfunction leads to the condition known as luteal phase deficiency (LPD) (166)