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Cyp15a, TPP , PR1 , and PR10 expression was upregulated and that of NF-YA1 downregulated in response to (h)GSH depletion in wild-type roots with nodules (Figure 7)
Probably due to the binding preference of APAP to the active site of each P450 isomer, it is oxidized via two pathways to form the toxic intermediate N-acetyl-p-benzoquinone imine (NAPQI) and the non-toxic catechol metabolite 3-hydroxy-APAP (3-OH-APAP) (Figure 1)
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