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doi: 10.3390/cimb44040096 366 PattersonETanHTTGroegerDAndrewsMBuckleyMMurphyEFet al

This is further supported by the inability of phlorizin to prevent glucose-induced GIP secretion in streptozotocin-treated, hyperglycemic mice, where complete blockage of GIP secretion is only achieved in these mice upon both phlorizin and K ATP channel activation (diazoxide) (Ogata et al., 2014)

[1] Tissue distribution of carnitine-biosynthetic enzymes in humans [edit] The tissue distribution of carnitine-biosynthetic enzymes in humans indicates TMLD to be active in the liver, heart, muscle, brain and highest in the kidneys