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In HEI-OC1 cells, overexpression of miR-34a resulted in autophagy flux impairment and cell activity suppression, which could be counteracted by miR-34a inhibition
For long-term studies, accounting for potential peptide degradation, even with proper storage, is a valid concern
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Incretin-based drugs and the incidence of endometrial cancer among people with type 2 diabetes: active comparator new-user design
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