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Department of Genetics and Clinical Immunology, National Institute of Tuberculosis and Lung Diseases, Warsaw, Poland Abstract Reactive oxygen and nitrogen species can be generated endogenously (by mitochondria, peroxisomes, and phagocytic cells) and exogenously (by pollutions, UV exposure, xenobiotic compounds, and cigarette smoke)
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Moreover, UF-1 inoculation also resulted in less degradation of endogenous MORF2 protein than that of the SsCm1 -17 mutant inoculation (Fig
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