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The potential for the therapeutic use of CETP inhibitors in humans was first suggested when it was discovered in 1985 that a small population of Japanese had an inborn error in the CETP gene leading to hyperalphalipoproteinemia and very high HDL levels
Glutathione-SR provides the direct substrate necessary for Glutathione S-transferases (GSTs) to perform their vital conjugation work
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normal homocysteine metabolism
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