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Flavin adenine dinucleotide (FAD) is a cofactor for MTHFR and the N-terminal and C-terminal domains of MTRR bind flavin mononucleotide and FAD, respectively (Leclerc et al
Mice deficient in AQP4 have been shown to survive longer than wild-type counterparts in in vivo brain edema models, highlighting its potential role in exacerbating BBB dysfunction [173]
Endotoxins can also cause oxidative stress, increasing the demand for glutathione to neutralize free radicals produced
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