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Research showed that activation of GPX4 was able to suppress inflammation, including cellular ROS level reduction and NF-B pathway suppression, and suggested boosting GPX4 activity could be considered a novel anti-inflammatory strategy in lipid peroxidation-associated diseases (Li et al., 2018)

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Studies examining fibroblast and endothelial cell systems show increased phosphorylation of focal adhesion kinase and paxillin, two proteins central to cytoskeletal organization and cell adhesion signaling

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