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Some studies have identified a correlation between T2DM and oxidative damage or antioxidant status [307,308,309]
While the liver plays a significant role in systemic glutathione metabolism, the central nervous system (CNS) maintains its own tightly regulated GSH pool, independent of peripheral control (3-4)
It is postulated that glutathione inhibits tyrosinase through three primary mechanisms: direct antioxidant activity, chelation of the copper site within tyrosinase via its thiol group, and disruption of tyrosinases cellular transport to pre-melanosomes
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