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To mitigate this drawback, the pharmacological administration of GIP, primarily known for its insulinotropic and mild appetite-suppressing properties [7], is also suggested to have an anti-emetic effect, potentially allowing GIP co-agonism to increase tolerability to the GLP-1 component [8]
Copy number profiling revealed none of the changes reported in the literature (Figure 4B) (19), and no copy number alteration was observed at the PRKAR1A locus
It is about preserving functional independence
The dangers come from how the treatment is delivered in the unregulated market
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