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Although tamoxifen and its metabolites (i.e., 4-hydroxytamoxifen) can cross the blood-brain barrier, these compounds have reduced bioavailability in the brain compared to other tissues 34
Lipid panel shows cholesterol impact
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removes ROS through superoxide dismutase activity, regulates the GPX4/SLC7a11 axis and Nrf2/HO-1 pathway to inhibit ferroptosis, forming a triple renal protective mechanism of ROS removal-ferroptosis inhibition-inflammation relief [77]
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