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Modulation of the GLP-1 - K ATP pathway by AMP kinase, NO and ACh The ability of GLP-1 released by RPc to relax cardiac pericytes and increase coronary blood flow presumably depends on the activation of K ATP channels in the surface membrane of the pericytes, which will hyperpolarise the pericytes and decrease voltage-gated Ca 2+ entry
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Some preclinical studies suggest expression on certain immune cells, though human evidence is variable
Figure 1 The prevalence of hsCRP levels 2 mg/L This study found (Figure 2) that 24.4% (21/86) of patients on the DTG-based regimen and 44.2% (38/86) on the ATV/r-based regimen have increased hsCRP levels 2 mg/L
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