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This, in turn, increases the numbers of L-cells, other intestinal secretory lineages, and GLP-1 secretion in both intestinal organoids and mice fed with normal chow and high-fat diet

Systematic integration of quantitative and qualitative information into the framework of an adverse outcome pathway revealed the causal involvement of oxidative stress in liver pathologies such as drug-induced liver injury, alcoholic liver disease, cholestatic liver injury, and nonalcoholic fatty liver disease (Kuijper et al

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